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Updated: Jan 7, 2026

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改造了线粒体和突触通路之间的GM1交叉路口,以改善ALS病理

Federica Pilotto1,2, Tristan Dellazizzo Toth3,4, Silvano Bond3,4

  • 1Institut Neuromyogène, Pathophysiology and genetics of the neuron and muscle, Inserm U1315, CNRS, Université Claude Bernard Lyon I, Lyon, France.

Advanced science (Weinheim, Baden-Wurttemberg, Germany)
|January 5, 2026
PubMed
概括

No abstract available in PubMed .

关键词:
美国ER与线粒体的接触其他SOD1 - G93A 其他肌缩侧面硬化症 (ALS)线粒体功能障碍纳米脂体配方蛋白质组学突触功能障碍

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