在高度城市化地区寻找食物的猫经历了激素和能量代谢的干扰
Coralie Turquois1, Marc J Mazerolle2, Sébastien Sauvé3
1Centre de recherche en toxicologie et santé de l'environnement (TOXEN), Département des sciences biologiques, Université du Québec à Montréal, Montréal, QC, Canada.
Environment international
|January 7, 2026
概括
城市鸟类暴露在制阻燃剂 (HFR) 和和多基物质 (PFAS) 中,对调节能量代谢的荷尔蒙产生性别特异性干扰,影响它们的健康和繁殖.
科学领域:
- 环境毒理学环境毒理学
- 野生动物生态毒理学
- 内分泌学 在内分泌学.
背景情况:
- 城市鸟类表现出高水平的制阻燃剂 (HFRs) 和和多基物质 (PFAS) 的血水平.
- 暴露于有机素可以破坏激素调节和能量代谢,可能影响鸟类的繁殖和健康.
- 关于HFRs和PFAS对城市野生动物能量代谢激素的特定影响的数据有限.
研究的目的:
- 为了研究HFRs和PFAS以及关键激素 (甲状腺,葡萄糖皮质激素) 和化环嘴海中的能量代谢标记物之间的血度之间的关系.
- 为了确定食息地使用是否影响有机素暴露水平.
- 为了评估与能量代谢相关的HFR和PFAS暴露的性别特异反应.
主要方法:
- 从筑巢的环嘴海 (Larus delawarensis) 的血样本中分析了HFR和PFAS度.
- 测量了甲状腺激素 (T3),葡萄糖皮质激素 (皮质) 和能量代谢标记物 (β-基酸盐).
- 在蒙特利尔地区,海的食活动被追踪了三年.
主要成果:
- 血HFR和PFAS度与食息地使用没有相关性,这表明城市来源广泛存在.
- 在有机素水平和代谢标志物之间观察到显著的性别相关联.
- PFAS暴露与男性降低β-基酸盐和女性增加水平有关;HFR与男性增加T3和β-基酸盐和女性增加皮质有关.
结论:
- 城市环境中的环海暴露于HFR和PFAS,导致性别特异的荷尔蒙和代谢干扰.
- 这些有机素诱导的能量代谢荷尔蒙的干扰可能对城市鸟类种群的健康和繁殖成功产生不利影响.
- 这些发现突出了城市生态系统中广泛传播的化学污染物对野生动物健康的潜在风险.
相关概念视频
Optimal Foraging
13.6K
How animals obtain and eat their food is called foraging behavior. Foraging can include searching for plants and hunting for prey and depends on the species and environment.
13.6K
Hormones Regulating Blood Glucose
6.4K
Insulin is released by beta cells of the pancreas when blood glucose levels are high. It facilitates glucose absorption and utilization in insulin-dependent cells with insulin receptors on their plasma membranes. Insulin promotes glucose uptake by increasing the number of glucose transport proteins in the cell membrane, allowing glucose to enter the cell. As a result, glucose utilization and ATP production are enhanced.
In addition to accelerating glucose uptake and utilization, insulin has...
In addition to accelerating glucose uptake and utilization, insulin has...
6.4K
Overview of Carbohydrate Metabolism
3.2K
Carbohydrate metabolism is a fundamental biochemical process that ensures a constant supply of energy to living cells. The most important carbohydrate is glucose, which can be broken down via glycolysis to enter into the Krebs cycle and eventually lead to the production of ATP through oxidative phosphorylation.
Glucose transport into cells is facilitated by a family of transport proteins called GLUT (Glucose Transporters). GLUT4 is the primary glucose transporter for insulin-stimulated glucose...
Glucose transport into cells is facilitated by a family of transport proteins called GLUT (Glucose Transporters). GLUT4 is the primary glucose transporter for insulin-stimulated glucose...
3.2K
Predator-Prey Interactions
21.0K
Predators consume prey for energy. Predators that acquire prey and prey that avoid predation both increase their chances of survival and reproduction (i.e., fitness). Routine predator-prey interactions elicit mutual adaptations that improve predator offenses, such as claws, teeth, and speed, as well as prey defenses, including crypsis, aposematism, and mimicry. Thus, predator-prey interactions resemble an evolutionary arms race.
21.0K
Metabolic States of the Body: Fasting and Starvation
2.7K
During the initial hours of fasting, the body uses up its glycogen stores as an energy source. Once these glycogen reserves are depleted, the body begins breaking down stored triglycerides and structural proteins. During this stage, glycerol becomes a key substrate for gluconeogenesis, while free fatty acids undergo beta-oxidation to provide energy for tissues, such as skeletal muscle. In the fasting state, the body spares protein breakdown as much as possible to conserve muscle and structural...
2.7K


