微RNA156及其向的SPL基因与光周期,白色化和吉伯雷林通路相互作用,调节小麦出发时间
Qiujie Liu1,2, Lili Zhang1,3, Zhicheng Zhou1
1University of California, Davis, California, 95616, USA.
The Plant journal : for cell and molecular biology
|January 11, 2026
概括
通过改变microRNA156和SQUAMOSA PROMOTER BINDING PROTEIN-LIKE基因来操纵小麦的年龄路径,可以加速转向时间. 这为改善小麦适应各种环境提供了新的遗传资源.
科学领域:
- 植物生物学 植物生物学
- 农作物科学 农作物科学
- 遗传学 是一个遗传学.
背景情况:
- 定位时间对于作物适应和生产率至关重要.
- 内生衰老途径在小麦出货时间中的作用尚未完全理解.
- 与光周期和白色化通路的相互作用是开花时间调节的关键.
研究的目的:
- 描述内源性年龄途径对四状小麦的行程时间的影响.
- 为了研究年龄,光周期和本地化途径之间的相互作用.
- 为了确定细调小麦出货时间的遗传资源.
主要方法:
- 微RNA156水平和SQUAMOSA PROMOTER BINDING PROTEIN-LIKE (SPL) 基因表达的表征. 这是一个很好的方法.
- 对促进开花 (例如VRN1,FUL2,FT1) 和抑制 (例如AP2L1,VRN2) 基因的分析.
- 使用遗传方法研究SPL,SQUAMOSA和DELLA蛋白质之间的相互作用.
主要成果:
- 减少miR156或增加SPL3/4/13表达加速方向时间,特别是在不理想的条件下.
- 以前的标题与miR172上调和特定的开花基因表达变化相关.
- 涉及DELLA蛋白质的复杂相互作用调节SPL活动,创建一个积极的反循环加速方向.
结论:
- 内生衰老路径,特别是miR156和SPL基因,显著影响小麦出发时间.
- SPL,SQUAMOSA和DELLA蛋白之间的相互作用形成了一个控制开花时间的调节网络.
- 开发出优势的miR156抗性SPL基因 (rSPL3,rSPL4,rSPL13) 为增强小麦适应提供了宝贵的工具.
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