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在糖尿病男性小鼠中改善勃起功能,通过拯救脱基化FBP1来减少洞穴乳酸

Ming Xiao1,2,3,4, Wanyang Guo1,2,3,4, Ruijiang Zeng1,2,3,4

  • 1Department of Urology, The Second Xiangya Hospital, Central South University, Changsha, Hunan, China.

Nature communications
|January 15, 2026
PubMed
概括

糖尿病引起的勃起功能障碍 (DMED) 是由雄性小鼠的乳酸积累驱动的. 通过mRNA疗法恢复果糖-1,6-双酸酶1 (Fbp1) 功能,改善了勃起功能.

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