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抗糖尿病DPP4抑制剂通过巨细胞核脉细胞交叉干线减轻椎间盘退化

Chen-Cheng Zhou1, Cheng-Long Xie1, Xiang-Cheng Zhang1

  • 1Key Laboratory of Orthopaedics of Zhejiang Province, Department of Orthopaedics, The Second Affiliated Hospital and Yuying Children's Hospital of Wenzhou Medical University, Wenzhou, Zhejiang, China.

British journal of pharmacology
|January 16, 2026
PubMed
概括

双基化酶4 (DPP4) 驱动椎间盘退化 (IVDD). 使用西塔利普丁抑制DPP4,通过调节巨细胞核脉细胞相互作用和炎症来减少IVDD的进展.

关键词:
DPP4 DPP4 DPP4 DPP4 DPP4 DPP4 DPP4 DPP4 DPP4 DPP4 DPP4IVDDDD IVDD 是一个很好的方法.门德尔的随机化它的核是脉冲的脉冲.西塔格利普丁 (Sitagliptin) 的使用情况.

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Dipeptidyl peptidase 4 (DPP-4) is a serine protease widely distributed in the body. It's involved in the inactivation of GLP-1 and GIP hormones, which are crucial for insulin regulation. DPP-4 inhibitors, such as sitagliptin (Januvia), saxagliptin (Onglyza), linagliptin (Tradjenta), alogliptin (Nesina), and vildagliptin (Galvus), help increase the proportion of active GLP-1, enhancing insulin secretion. These inhibitors work by competitively binding to DPP-4. This binding causes a...
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