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Regulation of Food Intake01:30

Regulation of Food Intake

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Short-term regulation of food intake primarily involves neural signals from the gastrointestinal (GI) tract, blood nutrient levels, and GI tract hormones. Communication between the gut and brain via vagal nerve fibers plays a significant role in evaluating the contents of the gut. Clinical studies have shown that protein ingestion produces a more prolonged response in these nerve fibers compared to an equivalent amount of glucose. Additionally, the activation of stretch receptors caused by GI...
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孕产妇的阿片类药物降低了阿迪波涅丁受体信号的调节,并改变了后代的生长:试点研究.

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    母亲阿片类药物使用障碍 (OUD) 可能会降低新生儿的阿迪波涅丁信号传递,可能导致异常生长和心脏代谢风险. 这项研究在暴露于阿片类药物的婴儿中发现了较低的ADIPOR1表达,这表明代谢编程发生了改变.

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    科学领域:

    • 新生儿健康 新生儿健康
    • 内分泌学 在内分泌学.
    • 公共卫生 公共卫生

    背景情况:

    • 成年人的阿片类药物使用障碍 (OUD) 与心脏代谢性疾病通过减少氨酸的作用有关.
    • 怀孕期间母亲使用的OUD可能会影响新生儿的生长,并导致成年人发病的疾病.
    • 孕产妇OUD对新生儿皮素水平的影响尚未研究.

    研究的目的:

    • 调查母体OUD对新生儿皮素水平的影响 (主要结局).
    • 检查母亲OUD对新生儿生长和身体组成的影响 (次要结果).
    • 为了比较暴露于阿片类药物和未暴露的新生儿之间的唾液腺素素受体1 (ADIPOR1) 表达.

    主要方法:

    • 一项前性观察试点研究,比较了35名暴露于阿片类药物的新生儿和32名未暴露的新生儿 (妊娠≥34周).
    • 测量了唾液ADIPOR1表达,人体测量和身体成分 (脂肪和无脂肪质量).
    • 根据暴露和性别分层的数据;使用Student的t-test进行分析 (p<0.05显著性).

    主要成果:

    • 暴露于阿片类药物的新生儿显示ADIPOR1表达显著降低 (0.27倍,p<0.01),特别是那些接受药物治疗的新生儿 (0.048倍,p<0.001).
    • 暴露于阿片类药物的新生儿具有较小的人类测量量 (2915g vs 3209g,p=0.02),但可比较的脂肪度 (8.60% vs 8.53%,p=0.95).
    • 在暴露于阿片类药物的组中观察到较低的母乳摄入量 (25.7%与71.9%,p<0.01).

    结论:

    • 母亲的OUD与新生儿的ADIPOR1表达减少有关.
    • 异常生长和后代的肥胖可能与通过阿迪波内克丁信号通路与母亲的OUD有关.
    • 暴露于母体OUD的新生儿可能对心脏代谢风险的倾向性增加.