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Updated: Jan 21, 2026

Radial Mobility and Cytotoxic Function of Retroviral Replicating Vector Transduced, Non-adherent Alloresponsive T Lymphocytes
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Radial Mobility and Cytotoxic Function of Retroviral Replicating Vector Transduced, Non-adherent Alloresponsive T Lymphocytes

Published on: February 11, 2015

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IL-6通过重编程髓状细胞和损害细胞毒性淋巴细胞功能来驱动NSCLC中的化疗免疫疗法抵抗性

Yaning Yang1, Chengming Liu2, Lu Yang3

  • 1Department of Medical Oncology, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100021, China; Centre de Recherche des Cordeliers, Université Paris Cité, Sorbonne Université, Equipe labellisée par la Ligue contre le cancer, Inserm U1138, Paris, 75006, France; Metabolomics and Cell Biology Platforms, Gustave Roussy Institut, Villejuif, 94800, France.

Cancer letters
|January 19, 2026
PubMed
概括

高基线介质素-6 (IL-6) 水平预测了晚期非小细胞肺癌 (NSCLC) 患者接受化疗免疫治疗的不良结果. 升高的IL-6促进了免疫抑制性瘤微环境,导致对治疗的抗性.

关键词:
在IL-6的研究中,IL-6被认为是IL-6的基因.化学免疫疗法 化学免疫疗法免疫抑制性瘤微环境非小细胞肺癌的肺癌.电阻的阻力可以

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