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Updated: Jan 22, 2026

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在过去的十年里,对动脉样硬化的自研究进行了图书统计分析

Wen-Qing Xu¹, Ying-Xi Yang², Yue Ma³

¹Heart Center, Guilin People's Hospital, Guangxi, China.

|January 21, 2026

概括

自在动脉样硬化中起着关键作用,影响血管细胞和斑块稳定性. 了解其机制为预防和治疗心血管疾病提供了新的治疗点.

科学领域:

心血管研究研究心血管研究
细胞生物学细胞生物学
生物医学科学生物医学科学

背景情况:

动脉样硬化 (AS) 是心血管疾病的主要危险因素,包括失脂症,血栓形成和炎症.
自是一种细胞分解受损元件的过程,对于维持平衡至关重要.
自在AS病原发生中的作用,特别是在内皮细胞,巨细胞和VSMC中,影响炎症和脂质代谢,但机制复杂.

研究的目的:

从2014-2024年分析动脉样硬化的自的研究格局.
识别出版趋势,主要贡献者和该领域的研究热点.
为未来的自和动脉样硬化研究方向提供见解.

主要方法:

使用CiteSpace和R-Bibliometrix工具进行图书统计分析.
检查出版趋势,国家/机构贡献和期刊发行情况.
对关键词的同时出现和共同引用网络的分析.

主要成果:

关于动脉样硬化中自的研究显著增长,最近加速.
研究重点是自在血管细胞 (内皮细胞,巨细胞,VSMCs),炎症,氧化应激和脂质代谢中的作用.
适度的自支持血管细胞生存和斑块稳定性;过度的自可能促进细胞死亡和AS进展.

结论:

自对于线粒体质量控制和缓解AS中的斑块形成至关重要.
在血管细胞中阐明自的调节机制可以揭示AS的新型治疗点.
图书统计分析突出了研究趋势,指导了未来对自和动脉样硬化的研究.

关键词:

动脉样硬化动脉样硬化自自是自的过程.文献计量分析的分析线粒体质量控制的质量控制血管细胞是血管细胞.

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