骨髓血管结构的重塑是由venetoclax和azacitidine损伤引起的

Steven Ngo1, Giuseppe Alessandro D'Agostino2, Despoina Papazoglou1

  • 1The Francis Crick Institute, London, United Kingdom.

Blood
|January 29, 2026
PubMed
概括

威尼托克拉克斯和阿扎西提丁 (ven/aza) 疗法通过耗尽阴侧内皮细胞损害骨髓微环境. 这损害了造血干细胞的回归和移植,强调了内皮细胞完整性的重要性.

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