IL-6作为IFIT2-贫乏口腔状细胞癌中骨侵入的驱动因素

Kuo-Chu Lai1,2, Chia-Hsun Hsieh2, Chen-Hsuan Wang3

  • 1Department of Physiology and Pharmacology, College of Medicine, Chang Gung University, Taoyuan City, 33302, Taiwan.

概括

干扰素诱导的蛋白质与四基重复2 (IFIT2) 枯竭在口腔癌促进通过增加Interleukin-6 (IL-6) 信号通过骨侵袭. 准IL-6/IFIT2轴可能会抑制口腔状细胞癌的进展.

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