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Updated: Feb 7, 2026

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在博雷佐米布诱导的神经病变中,皮肤神经中TrkA的丰度增加

Yuying Jin1, Nadine Cebulla1, Daniel Schirmer1

  • 1Department of Neurology, University Hospital of Würzburg, Würzburg, Germany.

Brain pathology (Zurich, Switzerland)
|February 5, 2026
PubMed
概括

多发性骨髓瘤患者的博特佐米布治疗通过改变TrkA信号传递和神经血管相互作用而导致外周神经病变. 这项研究显示,TrkA蛋白在存活的神经纤维中的增加以及皮肤血管化的变化.

关键词:
化疗诱导的神经病变基于深度学习的定量分析.分子生物标志物分子生物标志物

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