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Updated: Jul 3, 2026

Quantitative 3D In Silico Modeling (q3DISM) of Cerebral Amyloid-beta Phagocytosis in Rodent Models of Alzheimer's Disease
09:33

Quantitative 3D In Silico Modeling (q3DISM) of Cerebral Amyloid-beta Phagocytosis in Rodent Models of Alzheimer's Disease

Published on: December 26, 2016

老龄化的分子机制如何驱动阿尔茨海默病的病理学

Junkai Chang1, Tingting Liu1, Xiangshu Cheng1

  • 1Center for Translational Neurourology, Huaihe Hospital of Henan University, Institute for Brain Sciences Research, School of Life Sciences, Henan University, Kaifeng 475004, China.

Cellular signalling
|February 12, 2026
PubMed
概括

老龄化显著恶化阿尔茨海默病 (AD) 通过促进粉样β和病理和神经炎症. 针对衰老机制为AD干预提供了新的治疗策略.

关键词:
衰老的衰老 衰老的衰老阿尔茨海默病的疾病阿尔茨海默病的疾病.干预策略 干预策略病理机制 病理机制 病理机制

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