介质单心卡斯特曼病被误诊为胃肠神经内分泌瘤
Gordon Hua1, David Barker2, Xuemei Wu2
1Surgery, Drexel University College of Medicine, Philadelphia, Pennsylvania, USA ghh34@drexel.edu.
BMJ case reports
|February 19, 2026
概括
卡斯特勒曼是个好人.
科学领域:
- 在瘤学瘤学.
- 病理学 病理学 病理学
- 放射学 放射学是一门学科.
背景情况:
- 卡斯特曼病是一种罕见的淋巴增殖性疾病,具有不同的临床表现.
- 症状可以从无症状到全身症状,如发烧和疲劳等.
研究的目的:
- 报告一个独特的卡斯特曼病病例,呈现为高血管腹部质量.
- 突出诊断挑战和多学科瘤委员会审查的重要性.
主要方法:
- 一个30多岁的男性患者的病例报告呈现出非特异性症状.
- 诊断工作包括CT,-68 DOTATATE PET和内镜超声波活检.
- 腹部质量的手术切除,随后进行了他的病理学分析.
主要成果:
- 手术前的成像显示出高血管质量和神经内分泌瘤.
- 最初的活检显示了一个良性反应性淋巴结.
- 组织病理学证实了质血管切除后的卡斯特曼病.
结论:
- 卡斯特曼病可以模仿其他腹部病理,包括神经内分泌瘤.
- 多学科评估对于精确诊断和治疗罕见疾病至关重要.
- 手术切除是治愈性的,患者康复良好,并接受密切监测.
相关概念视频
Other Disorders of Digestive System
1.7K
The gastrointestinal tract is susceptible to various disorders. If the lower esophageal sphincter is damaged, stomach acid can flow back into the esophagus, causing irritation and inflammation of the lining. This condition is called gastroesophageal reflux disease (known as heartburn) and may cause chest pain and difficulty swallowing. In the stomach, prolonged use of nonsteroidal anti-inflammatory drugs like aspirin, chronic alcohol consumption, bacterial infections such as Helicobacter...
1.7K
Gastritis-I: Introduction and Types
3.7K
Gastritis, defined by the inflammation or irritation of the stomach lining or gastric mucosa, manifests in several distinct forms: acute, chronic, reactive, and a specific subtype known as autoimmune metaplastic atrophic gastritis.
Acute gastritis presents as a sudden inflammation triggered by various stressors to the stomach lining, such as exposure to corrosive agents, local irritants like aspirin and other NSAIDs, alcohol consumption, radiation therapy, physical trauma, severe burns, sepsis,...
Acute gastritis presents as a sudden inflammation triggered by various stressors to the stomach lining, such as exposure to corrosive agents, local irritants like aspirin and other NSAIDs, alcohol consumption, radiation therapy, physical trauma, severe burns, sepsis,...
3.7K
Gastritis-II: Pathophysiology
1.9K
Gastritis is marked by disruption of the mucosal barrier that usually protects the stomach tissue from digestive juices and manifests in acute and chronic forms.
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
1.9K
Gastritis III: Clinical Manifestations and Management
1.9K
The clinical manifestations of gastritis can vary depending on the cause and type of gastritis, but some common symptoms may include the following.
Clinical manifestations of acute gastritis
The patient with acute gastritis may have a rapid onset of symptoms, such as epigastric pain or discomfort, dyspepsia, anorexia, hiccups, or nausea and vomiting, which can last from a few hours to a few days. Erosive or hemorrhagic gastritis may cause bleeding, which may manifest as blood in vomit or as...
Clinical manifestations of acute gastritis
The patient with acute gastritis may have a rapid onset of symptoms, such as epigastric pain or discomfort, dyspepsia, anorexia, hiccups, or nausea and vomiting, which can last from a few hours to a few days. Erosive or hemorrhagic gastritis may cause bleeding, which may manifest as blood in vomit or as...
1.9K
Esophageal Achalasia
45
Esophageal achalasia is a chronic neurogenic disorder characterized by impaired relaxation of the lower esophageal sphincter (LES) and absent or ineffective peristalsis in the distal esophagus. This leads to a functional obstruction without a physical blockage, despite significant disruption of esophageal motility.EtiologyAchalasia is caused by degeneration of the myenteric (Auerbach's) plexus, specifically the loss of inhibitory ganglion cells that produce vasoactive intestinal peptide...
45
Gastritis II: Pathophysiology
52
The pathophysiology of gastritis begins with the colonization of the stomach lining by Helicobacter pylori (H. pylori). This bacterium spreads mainly via the oral-oral route through saliva or shared utensils, and can also be transmitted in overcrowded or unhygienic environments through contaminated water, despite its brief survival outside the body.ColonizationOnce ingested, H. pylori enters the stomach and begins colonization by navigating through the mucus layer lining the stomach wall. It...
52


