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Updated: May 8, 2026

Exogenous Administration of Microsomes-associated Alpha-synuclein Aggregates to Primary Neurons As a Powerful Cell Model of Fibrils Formation
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Published on: June 26, 2018

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海马CA2中的微质反应与高级神经元α-synucleinopathy有关

Esteban Luna, Katheryn A Q Cousins, Sheina Emrani

    bioRxiv : the preprint server for biology
    |February 23, 2026
    PubMed
    概括

    莱维体疾病涉及α-synuclein传播,与海马中的微质反应相关. 这项研究表明,Lewy体疾病中的微质变化与病理传播和认知衰退相关,有助于疾病的分期.

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    Alzheimer Disease ll: Pathophysiology

    Alzheimer disease involves structural changes in the brain that begin long before symptoms appear. The most distinctive features are extracellular neuritic plaques and intracellular neurofibrillary tangles.Neuritic plaques form in the cerebral cortex and around blood vessels. These plaques contain a dense core of beta-amyloid (Aβ)—a toxic protein fragment that clumps outside neurons. The core is surrounded by damaged neuronal extensions, as well as reactive astrocytes and microglia. Abnormal...