TNFR2信号传递促进单细胞-MDSC分化和免疫抑制媒介的产生

Yuta Tsuji1, Masaki Inoue1, Tomoya Okuda1

  • 1The Faculty of Pharmaceutical Sciences, Laboratory of Cellular and Molecular Physiology, Kobe Gakuin University, Japan.

FEBS letters
|February 25, 2026
PubMed
概括

促进瘤的髓质衍生抑制细胞 (MDSCs) 依赖于TNF受体2型 (TNFR2) 信号传递. 向TNFR2可以调节MDSC功能,并通过减少免疫抑制来增强癌症治疗的潜力.

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