-17Ap53uPA

Durgesh Nandini Das1, Akarsha Balnadupete1, Rashmi Shetty1

  • 1Texas Lung Injury Institute, Department of Cellular and Molecular Biology, The University of Texas Health Science Center at Tyler, 11937 US Highway 271, Tyler, TX 75708, USA.

概括

干白素-17A (IL-17A) 通过促进细胞死亡和肌纤维细胞分化来驱动肺损伤和纤维化. 抑制IL-17A信号减少肺损伤和纤维修复,为肺纤维化提供潜在的治疗点.

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