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克洛扎通过调节胆能功能,氧化应激和老鼠的亡信号来缓解脂聚糖诱导的认知功能障碍

Vasudevan Mani1, Mohammed A Almatrafi1

  • 1Department of Pharmacology and Toxicology, College of Pharmacy, Qassim University, Buraydah 51452, Saudi Arabia.

Life (Basel, Switzerland)
|February 27, 2026
PubMed
概括

克洛扎 (CLZ) 治疗在神经毒性大鼠模型中改善了认知功能. 通过调节参与认知和细胞存活的关键分子通路,CLZ表现出神经保护作用.

关键词:
灭症 (apoptosis) 是一种死亡的过程.克洛扎类药物 克洛扎类药物认知功能 认知功能脂多糖类糖化物 (Lipopolysaccharide) 是一种脂多糖的化合物.神经退行症的神经退行症氧化应激是一种氧化应激.

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Antipsychotic drugs are a crucial treatment method for acute and chronic psychoses, bipolar illness, and behavioral disorders. The selection of these drugs depends on several factors, including the state of the disease, clinical judgment, possible drug interactions, and the patient's sensitivity to adverse effects. In immediate scenarios, such as delirium and dementia, short-term treatment with low doses of high-potency typical or atypical agents can effectively manage symptom exacerbation.
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