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Coronary Progenitor Cells and Soluble Biomarkers in Cardiovascular Prognosis after Coronary Angioplasty
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功能性自身抗体和冠状动脉微血管阻塞在STEMI:免疫机制和预后结果之间的翻译联系

Laura Iop1, Giovanni Civieri2, Giacomo Bernava1

  • 1Cardiovascular Disease Modeling and Regenerative Medicine, Department of Cardiac, Thoracic, Vascular Sciences and Public Health, University of Padua, Padua, Italy.

Frontiers in cardiovascular medicine
|March 13, 2026
PubMed
概括

针对angiotensin II type 1 (AT1R) 和endothelin-1 type A (ETAR) 受体的抗原性自身抗体在ST升高心肌梗塞 (STEMI) 患者中直接导致冠状动脉微血管阻塞 (CMVO). 受体阻塞可能为高风险的STEMI个体提供保护策略.

关键词:
一个AT1受体的AT1受体.接收器 ETA 接收器 ETA.ST升高心肌梗塞的心脏病发作这是自抗体的自抗体.这是冠状动脉微循环.内皮质功能障碍 在内皮质功能障碍受体对抗剂的受体对抗剂.

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