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Updated: Mar 15, 2026

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向 Prolyl 3-基酶 1 抑制胰腺癌的进展和巨细胞免疫力

Panzhu Bai1, Chengmin Liu1, Changying Fu2

  • 1Department of Systems Biology, School of Life Sciences, Guangdong Provincial Key Laboratory of Cell Microenvironment and Disease Research, Shenzhen Key Laboratory of Cell Microenvironment, Southern University of Science and Technology, Shenzhen, China.

Nature communications
|March 14, 2026
PubMed
概括

普3-基酶1 (P3H1) 通过调节波罗样酶1 (PLK1) 和β-catenin信号传递,驱动胰腺癌的进展. 抑制PLK1可以提高抗胰腺管道腺癌的化疗效果.

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The mammalian target of rapamycin or mTOR protein was discovered in 1994 due to its direct interaction with rapamycin. The protein gets its name from a yeast homolog called TOR. The mTOR protein complex in mammalian cells plays a major role in balancing anabolic processes such as the synthesis of proteins, lipids, and nucleotides and catabolic processes, such as autophagy in response to environmental cues, such as availability of nutrients and growth factors.
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