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Updated: May 5, 2026

Fractionation for Resolution of Soluble and Insoluble Huntingtin Species
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Published on: February 27, 2018

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亨廷顿病病理学的双轨模型:状缩调解了不适应性免疫失调

H Jeremy Bockholt1,2,3,4, Jordan D Clemsen1, Bradley T Baker1

  • 1Center for Translational Research in Neuroimaging and Data Science (TReNDS), Georgia State University, Atlanta, GA 30303, USA.

International journal of molecular sciences
|March 14, 2026
PubMed
概括

亨廷顿病 (HD) 病理包括两个轨道:轴突损伤 (NEFL) 和免疫失调 (TNFRSF8). 状缩驱动免疫系统的变化,为HD提供了新的生物标志物标.

关键词:
亨廷顿病就是亨廷顿病.努利萨 (Nulisa) 是一个无关紧要的人.生物标志物 生物标志物神经免疫轴的神经免疫轴神经退行症的神经退行症神经炎症是一种神经炎症.蛋白质组学 蛋白质组学条纹体的条纹体.

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