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血液溶解尿素综合征:基础科学

J L Moake¹

¹Baylor College of Medicine, Methodist Hospital, Houston, Texas 77030.

Lancet (London, England)

|February 12, 1994

概括

No abstract available in PubMed .

相关实验视频

血液溶解尿素综合征:基础科学

相关概念视频

Disulfide bond reduction of von Willebrand factor by ADAMTS-13.

Protein phosphatase 2B inhibition promotes the secretion of von Willebrand factor from endothelial cells.

Covalent regulation of ULVWF string formation and elongation on endothelial cells under flow conditions.

Novel ADAMTS-13 mutations in an adult with delayed onset thrombotic thrombocytopenic purpura.

Platelet-derived VWF-cleaving metalloprotease ADAMTS-13.

Platelets adhered to endothelial cell-bound ultra-large von Willebrand factor strings support leukocyte tethering and rolling under high shear stress.

相关实验视频

血液溶解尿素综合征:基础科学

相关概念视频

相关文章

Disulfide bond reduction of von Willebrand factor by ADAMTS-13.

Protein phosphatase 2B inhibition promotes the secretion of von Willebrand factor from endothelial cells.

Covalent regulation of ULVWF string formation and elongation on endothelial cells under flow conditions.

Novel ADAMTS-13 mutations in an adult with delayed onset thrombotic thrombocytopenic purpura.

Platelet-derived VWF-cleaving metalloprotease ADAMTS-13.

Platelets adhered to endothelial cell-bound ultra-large von Willebrand factor strings support leukocyte tethering and rolling under high shear stress.