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在NOS2 -/-接受者中减弱的急性心脏排斥与减少的亡相关

J Koglin1, D J Granville, T Glysing-Jensen

  • 1Cardiovascular Biology Laboratory, Harvard School of Public Health, Boston, Mass. 02115, USA.

Circulation
|February 17, 1999
PubMed
概括

氧化合成酶2 (NOS2) 通过诱导亡促进了急性心脏异位移植排斥. NOS2淘汰赛小鼠显示了移植细胞亡的减少和改善的结果,这表明NOS2是一个治疗点.

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