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Lessons to be learned: a case study approach--a case of temporal arteritis
1East Surrey Hospital, Redhill.
A patient experienced sudden vision loss due to anterior ischemic optic neuropathy (AION). Delayed treatment despite elevated erythrocyte sedimentation rate (ESR) resulted in total blindness in the affected eye.
Area of Science:
- Ophthalmology
- Neurology
- Rheumatology
Background:
- Anterior ischemic optic neuropathy (AION) is a common cause of sudden, painless vision loss in older adults.
- Giant cell arteritis (GCA) is an important treatable cause of AION, necessitating prompt diagnosis and management.
- Early detection and treatment of AION, particularly when associated with elevated inflammatory markers, are crucial for visual prognosis.
Observation:
- A 71-year-old male presented with acute, partial visual loss and an inferior visual field defect in the right eye.
- Clinical examination revealed reduced visual acuity, a relative afferent pupillary defect, and a pale, swollen optic disc, consistent with AION.
- An elevated erythrocyte sedimentation rate (ESR) of 75 mm/hr was noted 16 days after initial presentation.
Findings:
- The provisional diagnosis of AION was made promptly.
- Despite the elevated ESR, systemic steroid treatment was initiated late.
- The patient progressed to total blindness in the affected right eye due to delayed intervention.
Implications:
- This case highlights the critical importance of timely intervention in suspected AION, especially with elevated ESR.
- Failure to act promptly on inflammatory markers can lead to irreversible vision loss.
- Emphasizes the need for urgent management protocols for AION to prevent permanent visual impairment.
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