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Surface bound plasmin promotes migration of Streptococcus pneumoniae through reconstituted basement membranes

T Eberhard1, G Kronvall, M Ullberg

  • 1Division of Clinical Microbiology, Karolinska Hospital, Stockholm, Sweden.

Microbial Pathogenesis
|March 25, 1999
PubMed

Insights

Streptococcus pneumoniae uses plasminogen to penetrate basement membranes, a key step in bacterial meningitis pathogenesis. This adhesion and penetration mechanism is crucial for understanding how these bacteria cause infection.

Area of Science:

  • Microbiology
  • Pathogenesis
  • Biochemistry

Background:

  • Basement membrane penetration is vital for bacterial meningitis development.
  • Streptococcus pneumoniae is a primary cause of bacterial meningitis.

Purpose of the Study:

  • To investigate Streptococcus pneumoniae's ability to adhere to and penetrate basement membranes.
  • To explore the role of plasminogen in this process.

Main Methods:

  • Testing S. pneumoniae strain adherence to Matrigel and laminin.
  • Quantifying plasminogen binding sites using Scatchard analysis.
  • Assessing basement membrane penetration in a dual chamber model with and without plasminogen.

Main Results:

  • Most S. pneumoniae strains adhered to basement membrane components like laminin and collagen IV.
  • All tested strains bound significant amounts of plasminogen.
  • Penetration occurred within 3-4 hours only in the presence of plasminogen, not in its absence over 21 hours.

Conclusions:

  • Surface-associated plasminogen facilitates Streptococcus pneumoniae penetration of basement membranes.
  • This mechanism is a potential virulence factor in bacterial meningitis.

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