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Identification of three alternatively spliced variants of human CD28 mRNA

G Magistrelli1, P Jeannin, G Elson

  • 1Centre d'Immunologie Pierre Fabre, Saint Julien en Genevois, France.

Insights

Resting human T cells express soluble CD28 variants, alongside the membrane form, which may influence T cell responses. Activated T cells exclusively express the membrane CD28 mRNA.

Area of Science:

  • Immunology
  • Molecular Biology
  • Cell Biology

Background:

  • CD28 is crucial for T cell costimulation, with its gene organized into four exons.
  • Previous research identified a CD28 mRNA splice variant lacking most of exon 2.
  • The role of alternative splicing in CD28 expression in human T cells requires further investigation.

Purpose of the Study:

  • To identify and characterize novel alternatively spliced CD28 mRNA variants in non-stimulated human T cells.
  • To investigate the structural and potential functional implications of these variants.
  • To determine the expression patterns of CD28 variants in resting versus activated T cells.

Main Methods:

  • Analysis of alternatively spliced CD28 mRNA variants in human T cells using molecular techniques.
  • Sequencing and structural analysis of identified CD28 mRNA variants.
  • Comparison of CD28 mRNA expression in resting and activated T cells.

Main Results:

  • Three new alternatively spliced CD28 mRNA variants (CD28a-c) were identified in non-stimulated human T cells.
  • CD28a variant, similar in expression to full-length CD28, lacks exon 3, leading to a frameshift and loss of the transmembrane region, suggesting a soluble form.
  • CD28b and CD28c variants, expressed at lower levels, result from deletions involving exon 2 and exon 3.
  • Activated T cells exclusively express the membrane-bound CD28 mRNA.

Conclusions:

  • Resting human T cells constitutively express both membrane and soluble forms of CD28.
  • These soluble CD28 variants may play a distinct role in regulating T cell responses.
  • Differential expression of CD28 isoforms in resting and activated T cells highlights complex regulatory mechanisms in T cell activation.

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