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Transmissable spongiform encephalopathy (TSE) agents as crystalline forms of the prion protein (PrP) that multiply by allowing normal metabolic forms of PrP to join the crystal.

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A PBMC-based assay detects residual pro-inflammatory response in endoscopes after high-level disinfection undetected by ATP bioluminescence: a pilot study.

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Treatment of superficial pseudomonal infection with citric acid

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The key must fit: macrophages transport prion infection to the central nervous system and may determine the sites of infection within it.

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Alkaloidal glycosidase inhibitors (AGIs) as the cause of sporadic scrapie, and the potential treatment of both transmissible spongiform encephalopathies (TSEs) and human immunodeficiency virus (HIV) infection.

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