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Phosphorylation of tobacco mosaic virus movement protein abolishes its translation repressing ability

O V Karpova1, N P Rodionova, K I Ivanov

  • 1Department of Virology, Moscow State University, Moscow, 119899, Russia.

Virology
|August 12, 1999
PubMed

Insights

Phosphorylation of the Tobacco Mosaic Virus (TMV) movement protein (MP) converts nontranslatable MP-RNA complexes into a translatable and infectious form. This finding is crucial for understanding TMV infection and spread in plants.

Area of Science:

  • Plant virology
  • Molecular biology
  • Biochemistry

Background:

  • Ribonucleoprotein complexes (RNPs) of Tobacco Mosaic Virus (TMV) movement protein (MP) with TMV RNA are nontranslatable in vitro but infectious to intact plants.
  • It is hypothesized that MP-TMV RNA complexes convert to a translatable form during passage through plasmodesmata in planta.
  • The role of MP phosphorylation in modulating its translation-repressing ability requires investigation.

Purpose of the Study:

  • To investigate the role of TMV MP phosphorylation in modulating its translation-repressing ability.
  • To determine if phosphorylation converts nontranslatable MP-RNA complexes into a translatable and infectious form.

Main Methods:

  • Investigated the effect of TMV MP phosphorylation on RNP complex translatability and infectivity.
  • Examined phosphorylation before and after RNP complex formation.

Main Results:

  • Phosphorylation of TMV MP, irrespective of timing (before or after RNP complex formation), converted nontranslatable MP-RNA complexes.
  • The converted complexes became translatable in vitro and infectious to protoplasts and plants.

Conclusions:

  • TMV MP phosphorylation is a key mechanism for converting nontranslatable MP-RNA complexes into a translatable and infectious state.
  • This phosphorylation event is critical for TMV infection and spread within plant tissues.

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