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Serum soluble intercellular adhesion molecule 1 and interleukin 8 levels in familial Mediterranean fever

H Direskeneli1, H Ozdogan, C Korkmaz

  • 1Division of Rheumatology, Faculty of Medicine, Marmara University, Turkey.

The Journal of Rheumatology
|September 24, 1999
PubMed
Abstract

Insights

Familial Mediterranean fever (FMF) involves elevated levels of soluble intercellular adhesion molecule 1 (sICAM-1) and interleukin 8 (IL-8), indicating neutrophil activity and adhesion. These markers suggest ongoing subclinical inflammation even during remission in FMF patients.

Area of Science:

  • Immunology
  • Rheumatology
  • Genetics

Background:

  • Familial Mediterranean fever (FMF) is an autoinflammatory disease characterized by recurrent polyserositis and fever, with an unknown etiology.
  • Key mediators in leukocyte-endothelial adhesion and tissue accumulation include soluble intercellular adhesion molecule 1 (sICAM-1) and interleukin 8 (IL-8).

Purpose of the Study:

  • To investigate the levels of sICAM-1 and IL-8 in patients with FMF during both active attacks and remission periods.
  • To explore the potential role of these inflammatory markers in the pathogenesis and disease activity of FMF.

Main Methods:

  • sICAM-1 and IL-8 levels were quantified using commercial ELISA systems.
  • The study included 30 FMF patients (during attacks and remission) and compared them with 23 healthy and 26 disease controls.

Main Results:

  • Median sICAM-1 levels were significantly elevated in FMF patients during attacks and remission compared to healthy controls (p<0.0001 and p=0.002, respectively).
  • IL-8 levels were significantly higher during FMF attacks compared to healthy controls (p=0.009), but not during remission (p=0.7).
  • A significant positive correlation was found between sICAM-1 and IL-8 levels (r=0.33, p=0.01), as well as with acute phase reactants like ESR, CRP, and fibrinogen.

Conclusions:

  • Elevated sICAM-1 and IL-8 in FMF suggest increased neutrophil activity and adhesion.
  • The persistent elevation of sICAM-1 during remission indicates subclinical disease activity and ongoing inflammation in some FMF patients.

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