Biochemical expression of degenerative disorders associated with cognitive dysfunction

Decreased lysosomal subunit c-degrading activity in fibroblasts from patients with late infantile neuronal ceroid lipofuscinosis.

Neuropediatrics·1997

Specific delay in the degradation of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid lipofuscinosis is derived from cellular proteolytic dysfunction rather than structural alteration of subunit c.

Journal of neurochemistry·1996

New insight into lysosomal protein storage disease: delayed catabolism of ATP synthase subunit c in Batten disease.

Neurochemical research·1995

Abnormal degradative pathway of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid-lipofuscinosis (Batten disease).

American journal of medical genetics·1995

Specific delay of degradation of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid lipofuscinosis (Batten disease).

Journal of neurochemistry·1995

Defect of proteolysis of mitochondrial ATP synthase subunit C in neuronal ceroid lipofuscinosis.

Gerontology·1995

Cytokine circuits in brain. Implications for AIDS dementia complex.

Research publications - Association for Research in Nervous and Mental Disease·1994

HIV-related depression.

Research publications - Association for Research in Nervous and Mental Disease·1994

White Matter Functional Dysregulation in Amyotrophic Lateral Sclerosis: Machine Learning-Based Biomarkers and Transcriptomic Signatures.

Current neuropharmacology·2026

Combined bidirectional spinal cord stimulation for refractory neuropathic pelvic pain: a case report.

Frontiers in pain research (Lausanne, Switzerland)·2026

Neurovascular unit uncoupling in diabetic retinopathy: molecular mechanisms and stage-adapted therapeutic strategies.

Frontiers in endocrinology·2026

Late-onset ornithine transcarbamylase deficiency with neurological damage and serial brain multimodality monitoring: a case report.

Frontiers in medicine·2026

The influence of magnesium ions on the electrophysiological, analgesic, and BDNF-induced neuromodulation of morphine effects in diabetic rats.

Frontiers in pharmacology·2026

Risk factors of nasogastric tube removal failure in acute ischemic stroke patients: a 3-months follow-up cohort study at a single institute.

Frontiers in neurology·2026

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Biochemical expression of degenerative disorders associated with cognitive dysfunction

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Decreased lysosomal subunit c-degrading activity in fibroblasts from patients with late infantile neuronal ceroid lipofuscinosis.

Specific delay in the degradation of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid lipofuscinosis is derived from cellular proteolytic dysfunction rather than structural alteration of subunit c.

New insight into lysosomal protein storage disease: delayed catabolism of ATP synthase subunit c in Batten disease.

Abnormal degradative pathway of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid-lipofuscinosis (Batten disease).

Specific delay of degradation of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid lipofuscinosis (Batten disease).

Defect of proteolysis of mitochondrial ATP synthase subunit C in neuronal ceroid lipofuscinosis.

Cytokine circuits in brain. Implications for AIDS dementia complex.

HIV-related depression.

White Matter Functional Dysregulation in Amyotrophic Lateral Sclerosis: Machine Learning-Based Biomarkers and Transcriptomic Signatures.

Combined bidirectional spinal cord stimulation for refractory neuropathic pelvic pain: a case report.

Neurovascular unit uncoupling in diabetic retinopathy: molecular mechanisms and stage-adapted therapeutic strategies.

Late-onset ornithine transcarbamylase deficiency with neurological damage and serial brain multimodality monitoring: a case report.

The influence of magnesium ions on the electrophysiological, analgesic, and BDNF-induced neuromodulation of morphine effects in diabetic rats.

Risk factors of nasogastric tube removal failure in acute ischemic stroke patients: a 3-months follow-up cohort study at a single institute.

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Biochemical expression of degenerative disorders associated with cognitive dysfunction

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