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Clinical severity in CADASIL related to ultrastructural damage in white matter: in vivo study with diffusion tensor

H Chabriat1, S Pappata, C Poupon

  • 1Department of Neurology, CHU Lariboisière, Université Paris VII, France. chabriat@ccr.jussieu.fr

Stroke
|December 3, 1999
PubMed
Abstract

Insights

Diffusion tensor imaging (DTI) reveals significant white matter changes in Cerebral Autosomal-Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CADASIL). These microstructural alterations correlate with disease severity, suggesting DTI

Area of Science:

  • Neuroimaging
  • Neurology
  • Biomedical Engineering

Background:

  • Cerebral Autosomal-Dominant Arteriopathy with Subcortical Infarcts and Leukoencephalopathy (CADASIL) is a genetic disorder causing progressive subcortical ischemic strokes, dementia, and motor disability.
  • The clinical presentation and disease progression in CADASIL are highly variable.
  • The role of MRI-detected white matter abnormalities in the clinical severity of CADASIL is not well understood.

Purpose of the Study:

  • To investigate the relationship between white matter microstructural changes and clinical severity in CADASIL using diffusion tensor imaging (DTI).
  • To assess the sensitivity of DTI in detecting ultrastructural alterations in both lesional and non-lesional white matter in CADASIL patients.

Main Methods:

  • Diffusion tensor imaging (DTI) was performed on 16 symptomatic CADASIL patients and 10 age-matched controls.
  • Mean diffusivity and diffusion anisotropy were measured within T2-weighted image (T2WI) hyperintensities and normal-appearing white matter.
  • Measurements were taken on four slices at the centrum semiovale level.

Main Results:

  • DTI revealed a 60% increase in mean diffusivity and reduced anisotropy within T2WI hyperintensities in CADASIL patients compared to controls.
  • Similar, though less pronounced, diffusion changes were observed in normal-appearing white matter.
  • Increased diffusivity in T2WI lesions correlated with Mini-Mental State Examination and Rankin scale scores, with higher diffusivity in severely affected patients.

Conclusions:

  • DTI effectively detects significant ultrastructural changes in both lesional and normal-appearing white matter in CADASIL.
  • These diffusion changes, potentially reflecting neuronal loss and demyelination, correlate with clinical disability severity.
  • DTI shows promise as a tool for monitoring disease progression in CADASIL.

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