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Macrolide resistance mechanisms and expression of phenotypes among Streptococcus pneumoniae circulating in Italy

A Marchese1, E Tonoli, E A Debbia

  • 1Institute of Microbiology, University of Genoa, Italy.

Insights

Macrolide resistance in Streptococcus pneumoniae is rising in Italy. Most resistant strains carry the MLS(B) mechanism, suggesting a widespread, highly transmissible clone is circulating.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Epidemiology

Background:

  • Macrolide resistance in Streptococcus pneumoniae (pneumococci) has been increasing in Italy.
  • From 1993 to 1998, resistance rates rose from 6% to 31.7%.

Purpose of the Study:

  • To characterize the phenotypic and genotypic mechanisms of erythromycin resistance in Streptococcus pneumoniae isolates from Italy.
  • To investigate the genetic relatedness and spread of resistant pneumococcal strains.

Main Methods:

  • Phenotypic characterization of erythromycin resistance.
  • Genotypic analysis using Polymerase Chain Reaction (PCR) to detect resistance genes (ermB, mefE).
  • Pulsed-field gel electrophoresis (PFGE) of chromosomal DNA to assess strain relatedness.

Main Results:

  • 161 erythromycin-resistant Streptococcus pneumoniae isolates were analyzed.
  • Approximately 90% exhibited a constitutive MLS(B) resistance mechanism.
  • PCR confirmed ermB genes in MLS(B) strains and mefE genes in M phenotype strains.
  • PFGE revealed a dominant restriction profile in 51 isolates, indicating a common, widespread clone.

Conclusions:

  • A significant increase in macrolide-resistant pneumococci has been observed in Italy.
  • The constitutive MLS(B) mechanism, mediated by the ermB gene, is the predominant resistance mechanism.
  • Evidence suggests the circulation of a highly transmissible clone of Streptococcus pneumoniae responsible for spreading macrolide resistance.

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