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Characterization and induction of aryl hydrocarbon (benzo(a)pyrene) hydroxylase in rabbit bone marrow

Research Communications in Chemical Pathology and Pharmacology
|October 1, 1976
PubMed

Insights

Benzo(a)pyrene hydroxylase activity in rabbit bone marrow, particularly in white cells, suggests a role for mixed-function oxygenases in hemopoietic toxicity. This enzyme activity was NADPH dependent and CO sensitive.

Area of Science:

  • Biochemistry
  • Toxicology
  • Hematology

Background:

  • Bone marrow is a critical site for hematopoiesis.
  • Mixed-function oxygenases (MFOs) metabolize xenobiotics.
  • Cytochrome P-450 enzymes are key components of MFO systems.

Purpose of the Study:

  • To investigate Benzo(a)pyrene (BP) hydroxylase activity in rabbit bone marrow.
  • To characterize the properties and cellular localization of this activity.
  • To explore the potential role of bone marrow MFOs in hemopoietic toxicity.

Main Methods:

  • Enzyme assays were performed on bone marrow microsomes from control and 3-methylcholanthrene (3MC)-induced rabbits.
  • NADPH and carbon monoxide (CO) dependence were assessed.
  • Inhibition by 7,8-benzoflavone was evaluated.
  • Cellular localization within bone marrow was determined.

Main Results:

  • BP hydroxylase activity was detected in rabbit bone marrow microsomes.
  • The activity was NADPH dependent and CO sensitive, characteristic of cytochrome P-450.
  • 7,8-benzoflavone inhibited the activity, indicating involvement of the 3MC-inducible P-450 form.
  • Activity was primarily located in white blood cells and was higher than in human lymphocytes/monocytes.

Conclusions:

  • Rabbit bone marrow possesses significant BP hydroxylase activity, mediated by a 3MC-inducible cytochrome P-450.
  • This activity is predominantly found in white cells.
  • Bone marrow MFOs may play a role in causing hemopoietic toxicity.

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