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Differential hepatic lobar gene expression in offspring exposed to altered maternal dietary protein intake

J Zhang1, C D Byrne

  • 1Endocrine and Metabolism Unit, University of Southampton School of Medicine, Southampton General Hospital, Southampton SO16 6YD, United Kingdom.

Insights

Maternal low-protein diets reduce fibrinogen gene expression and protein in adult offspring. This effect is localized to the left liver lobe and linked to reduced glucocorticoid receptor function.

Area of Science:

  • Biochemistry
  • Developmental Biology
  • Nutritional Science

Background:

  • Elevated plasma fibrinogen is a risk factor for coronary heart disease.
  • Fibrinogen levels in adults correlate with reduced early growth parameters.
  • The liver's unique fetal blood supply creates differential stimuli between left and right lobes.

Purpose of the Study:

  • To investigate the impact of maternal low-protein (MLP) diet on fibrinogen gene expression in adult offspring.
  • To determine if MLP effects on fibrinogen are consistent between the left and right liver lobes.
  • To explore the underlying mechanisms involving glucocorticoid receptor function.

Main Methods:

  • Studied fibrinogen gene expression (mRNA copy numbers) in adult offspring of control and MLP dams.
  • Assessed fibrinogen concentration in liver lobes.
  • Analyzed glucocorticoid receptor binding affinity and mRNA levels in liver lobes.

Main Results:

  • MLP offspring showed reduced fibrinogen mRNA and protein specifically in the left liver lobe.
  • No significant effects were observed in the right liver lobe or in control offspring.
  • Reduced glucocorticoid receptor binding affinity and mRNA levels were found in the left liver lobe of MLP offspring.

Conclusions:

  • Maternal dietary protein restriction leads to reduced fibrinogen gene expression, protein, and glucocorticoid receptor function.
  • These effects are anatomically restricted to the left liver lobe in adult offspring.
  • Early-life nutrition significantly influences long-term gene expression and receptor function in a tissue-specific manner.

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