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The hypocretins are weak agonists at recombinant human orexin-1 and orexin-2 receptors

D Smart1, J C Jerman, S J Brough

  • 1Neuroscience Research, SmithKline Beecham Pharmaceuticals, New Frontiers Science Park, Third Avenue, Harlow, Essex CM19 5AW, UK. Darren_2_Smart@sbphrd.com

Insights

Hypocretins are weak agonists at orexin receptors. Orexin-A and orexin-B effectively activate orexin-1 (OX(1)) and orexin-2 (OX(2)) receptors, while hypocretins show minimal potency.

Area of Science:

  • Pharmacology
  • Neuroscience
  • Cell Biology

Background:

  • Orexin-like peptides, hypocretin-1 and hypocretin-2, are involved in regulating various physiological functions.
  • Orexin receptors (OX(1) and OX(2)) mediate the actions of orexin peptides.

Purpose of the Study:

  • To investigate the pharmacological profiles of hypocretin-1 and hypocretin-2 at OX(1) and OX(2) receptors.
  • To compare the potency of hypocretins with orexin-A and orexin-B.

Main Methods:

  • Utilized Chinese hamster ovary (CHO) cells stably expressing OX(1) or OX(2) receptors.
  • Measured intracellular calcium ([Ca(2+)](i)) increase using Fluo-3AM as an indicator of receptor activation.
  • Performed receptor desensitization experiments with orexin-A.

Main Results:

  • Orexin-A and orexin-B demonstrated significant potency at both OX(1) and OX(2) receptors (pEC(50) values ranging from 7.00 to 8.30).
  • Hypocretin-1 and hypocretin-2 exhibited markedly lower potency, particularly at OX(2) receptors (pEC(50) values around 5.3-5.4).
  • Hypocretins elicited only partial responses in CHO-OX(1) cells, even at high concentrations, and receptor desensitization experiments confirmed their weak agonistic activity.

Conclusions:

  • Hypocretins act as weak agonists at both orexin-1 and orexin-2 receptors.
  • Orexin-A and orexin-B are potent agonists, highlighting distinct pharmacological profiles compared to hypocretins.

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