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Apolipoprotein E allele distribution in trisomy 13, 18, and 21 conceptuses in a Hungarian population

B Nagy1, Z Bán, E Tóth-Pál

  • 1First Department of Obstetrics and Gynecology, Semmelweis University Medical School, Budapest, Hungary.

Insights

Apolipoprotein E (apoE) epsilon 4 allele showed a 3-fold increased odds for trisomy 18, but no significant association was found for trisomy 13 or 21. Further research is needed on apoE

Area of Science:

  • Genetics
  • Human Biology
  • Molecular Biology

Background:

  • Previous reports suggested a link between apolipoprotein E (apoE) epsilon 4 allele and Down syndrome.
  • The apolipoprotein E gene plays a role in lipid metabolism and has been implicated in various health conditions.

Purpose of the Study:

  • To investigate the prevalence of apolipoprotein E (apoE) alleles in fetuses with trisomy 13, 18, and 21.
  • To determine if specific apoE alleles are associated with an increased risk of these chromosomal abnormalities.

Main Methods:

  • Analysis of apolipoprotein E (apoE) alleles (epsilon 2, epsilon 3, epsilon 4) using polymerase chain reaction-restriction fragment length polymorphism.
  • Detection of trisomy status (13, 18, 21) via fluorescent polymerase chain reaction and conventional cytologic methods.
  • Comparison of allele frequencies between trisomic conceptuses and a healthy control group.

Main Results:

  • No significant difference in apolipoprotein E (apoE) allele distribution was observed for trisomy 21 fetuses compared to controls.
  • A statistically significant 3-fold increased odds of trisomy 18 was associated with the apolipoprotein E (apoE) epsilon 4 allele compared to the epsilon 3 allele.
  • No significant association was found between the apolipoprotein E (apoE) epsilon 4 allele and trisomy 13.

Conclusions:

  • The apolipoprotein E (apoE) epsilon 4 allele is significantly associated with trisomy 18, but not trisomy 13 or 21.
  • The observed frequencies of apolipoprotein E (apoE) epsilon 3 in Down syndrome patients and controls may support previous findings of lower atherosclerosis rates.
  • The precise role of apolipoprotein E (apoE) alleles in the etiology of trisomies requires further investigation.

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