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Relationship between benign prostatic hyperplasia and history of coronary artery disease in elderly men

K M Weisman1, G E Larijani, M R Goldstein

  • 1Department of Surgery, Taylor Hospital, Ridley Park, Pennsylvania, USA.

Pharmacotherapy
|April 20, 2000
PubMed

Insights

Elderly men with benign prostatic hyperplasia (BPH) showed a higher frequency of coronary artery disease (CAD). This suggests a potential link between androgen-dependent conditions like BPH and atherosclerosis development.

Area of Science:

  • Urology
  • Cardiology
  • Andrology

Background:

  • Benign prostatic hyperplasia (BPH) is an androgen-dependent condition common in elderly men.
  • Coronary artery disease (CAD) is a significant health concern in aging populations.
  • The potential relationship between BPH and CAD has not been extensively studied.

Purpose of the Study:

  • To investigate the association between the presence of benign prostatic hyperplasia (BPH) and coronary artery disease (CAD) in elderly men.
  • To explore potential shared etiological factors, such as androgen dependency, in the development of both conditions.

Main Methods:

  • A retrospective chart review was conducted on 702 elderly men aged 65-80 years.
  • Data on coronary artery disease (CAD) history, risk factors, and serum prostate-specific antigen (PSA) levels were collected.
  • Exclusion criteria included conditions or treatments that could affect PSA levels or lipid profiles.

Main Results:

  • Serum prostate-specific antigen (PSA) levels positively correlated with prostatic volume in BPH.
  • Men with BPH (PSA > 1.0 microg/L) had a significantly higher frequency of CAD (29%) compared to men without BPH (PSA < 1.0 pg/L) (9%) (p<0.03).
  • No significant differences in other CAD risk factors like age, smoking, diabetes, or hypertension were observed between groups.

Conclusions:

  • Smooth muscle proliferation, potentially androgen-dependent, may play a role in the pathogenesis of both atherosclerosis and BPH.
  • Further prospective studies are warranted to explore the impact of antiandrogen therapies on atherosclerosis.
Abstract

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