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Role of nitric oxide on insulin induced seizures in mice

V K Bhargava1, S Balakrishnan

  • 1Department of Pharmacology, PIGIMER, Chandigarh.

Insights

Nitric oxide (NO) protects against seizures caused by acute hypoglycemia. Boosting NO levels with L-arginine prevented seizures, while inhibiting NO synthesis with L-NMMA worsened them.

Area of Science:

  • Neuroscience
  • Pharmacology
  • Biochemistry

Background:

  • Acute hypoglycemia can trigger seizures, a serious neurological complication.
  • Nitric oxide (NO) is a signaling molecule implicated in various brain functions.
  • Understanding NO's role in hypoglycemia-induced seizures is crucial for potential therapeutic interventions.

Purpose of the Study:

  • To investigate the role of nitric oxide (NO) in acute hypoglycemia-induced seizures.
  • To determine the effects of NO modulation (precursor and inhibitor) on seizure activity.

Main Methods:

  • Insulin was used to induce acute hypoglycemia and seizures in a mouse model.
  • The NO precursor, L-arginine, was administered at varying doses (150, 500, 750 mg/kg).
  • The NO synthase inhibitor, L-NMMA, was administered at varying doses (50, 100 mg/kg).
  • Seizure onset, duration, number, and mortality were recorded over a 2-hour period.

Main Results:

  • L-arginine demonstrated a dose-dependent protective effect against insulin-induced seizures.
  • L-NMMA potentiated the seizure-inducing effects of subconvulsive insulin doses.
  • NO modulation significantly impacted seizure parameters and mortality.

Conclusions:

  • Nitric oxide (NO) plays a significant protective role in mitigating acute hypoglycemia-induced seizures.
  • These findings suggest NO-based strategies could be beneficial for managing seizures associated with hypoglycemia.
  • The mechanism may involve the modulation of NMDA receptor activity.

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