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Updated: Aug 13, 2026

Isolation and Cannulation of Cerebral Parenchymal Arterioles
Published on: May 23, 2016
[Cerebral panthrombophlebitis in a paucisymptomatic patient]
H Blachère1, V Dousset, O Berger
1Service de Neuroradiologie, CHU de Bordeaux, Groupe Hospitalier Pellegrin-Tripode, Bordeaux.
A cerebral venous thrombosis case with mild symptoms showed a good prognosis after anticoagulation treatment. The underlying cause was identified as activated protein C resistance, highlighting effective treatment outcomes.
Area of Science:
- Neurology
- Vascular Medicine
- Hematology
Background:
- Cerebral venous thrombosis (CVT) often leads to severe outcomes and poor prognosis.
- Prompt diagnosis and treatment are crucial for managing CVT.
Observation:
- A 59-year-old patient presented with subtle symptoms despite extensive cerebral venous thrombosis affecting both superficial and deep venous systems.
- The patient's condition involved significant venous structure compromise.
Findings:
- Etiologic assessment identified activated protein C resistance as the underlying cause.
- Systemic anticoagulation therapy resulted in a prompt and positive clinical evolution.
- Complete repermeabilization of affected venous structures was observed.
Implications:
- This case suggests that even extensive cerebral venous thrombosis can have a favorable prognosis with appropriate anticoagulation.
- Activated protein C resistance should be considered in the etiological assessment of CVT.
- Early and effective anticoagulation is key to successful CVT management and venous recanalization.
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