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Supplemental intraoperative oxygen augments antimicrobial and proinflammatory responses of alveolar macrophages

N Kotani1, H Hashimoto, D I Sessler

  • 1Department of Anesthesiology, University of Hirosaki, Japan. nao@cc.hirosaki-u.ac.jp

Anesthesiology
|June 22, 2000
PubMed
Abstract

Insights

Intraoperative 100% oxygen improved immune cell function and boosted anti-inflammatory responses in patients during surgery. This suggests higher oxygen levels enhance the body's defense mechanisms during anesthesia.

Area of Science:

  • Anesthesiology
  • Immunology
  • Pulmonary Medicine

Background:

  • Investigating the effects of intraoperative 100% oxygen on pulmonary oxygenation and immune responses.
  • Testing the hypothesis that prolonged 100% oxygen exposure does not impair lung function.
  • Examining the impact of 100% oxygen on alveolar macrophage proinflammatory and antimicrobial activities.

Purpose of the Study:

  • To evaluate if 100% inspired oxygen during 8-hour surgery impairs pulmonary oxygenation.
  • To determine if intraoperative 100% oxygen augments proinflammatory and antimicrobial responses of alveolar macrophages.

Main Methods:

  • Patients received either 100% or 30% oxygen during propofol-fentanyl anesthesia.
  • Alveolar macrophages were collected via bronchoalveolar lavage at multiple time points.
  • Phagocytosis, microbicidal activity, and gene expression of cytokines (IL-1beta, IL-6, IL-8, IFN-gamma, TNF-alpha) were measured.

Main Results:

  • Gene expression for most proinflammatory cytokines increased in both groups over time.
  • 100% oxygen group showed 2-20 times greater expression of TNF-alpha, IL-8, IFN-gamma, IL-6, and IL-1beta compared to 30% oxygen.
  • Phagocytic and microbicidal activity decreased in both groups, but less significantly with 100% oxygen.

Conclusions:

  • Intraoperative 100% oxygen may improve phagocytic and microbicidal activity by augmenting proinflammatory cytokine expression.
  • Findings suggest that 100% oxygen enhances antimicrobial and proinflammatory responses in alveolar macrophages during surgery.

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