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Mycobacterium tuberculosis infection in pediatric liver transplant recipients

A Verma1, A Dhawan, J J Wade

  • 1Dulwich Public Health Laboratory and Medical Microbiology, King's College Hospital, London, UK. anil.dhawan@kcl.ac.uk

Insights

Tuberculosis (TB) affects 2.4% of pediatric liver transplant recipients, causing significant morbidity and mortality. Early screening and vigilant monitoring for isoniazid-induced hepatitis are crucial for effective management and improved outcomes in these vulnerable patients.

Area of Science:

  • Pediatric Hepatology
  • Transplant Infectious Diseases
  • Mycobacterial Infections

Background:

  • Liver transplantation in children is a complex procedure with potential for serious post-transplant complications.
  • Tuberculosis (TB) remains a global health challenge, posing a particular risk to immunocompromised populations, including pediatric transplant recipients.

Observation:

  • A retrospective review identified a 2.4% incidence of Mycobacterium tuberculosis infection in 254 pediatric liver transplant recipients between 1991 and 1998.
  • Common clinical presentations included cough, pyrexia, and poor appetite, with half of patients having normal chest radiographs.
  • Diagnostic methods involved Ziehl-Neelsen stain, M. tuberculosis PCR, Mantoux testing, and histopathology, with family screening aiding diagnosis in 4 cases.

Findings:

  • The median time to diagnosis was 8 months, with treatment durations ranging from 9 to 18 months.
  • Isoniazid-induced hepatitis occurred in two patients but resolved with dose adjustment.
  • Mortality was observed in two patients due to Klebsiella septicemia and pulmonary hemorrhage.

Implications:

  • Tuberculosis post-liver transplantation is associated with substantial morbidity and mortality.
  • Pretransplant screening for personal and family history of TB, along with patient and family screening, is recommended.
  • Standard anti-TB regimens are effective, but regular liver function monitoring is essential to manage drug-induced hepatotoxicity.
Abstract

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