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Human monocytes express CD163, which is upregulated by IL-10 and identical to p155

T H Sulahian1, P Högger, A E Wahner

  • 1Department of Physiology, Dartmouth Medical School, Lebanon, NH, 03756, USA.

Cytokine
|September 8, 2000
PubMed

Insights

CD163 is highly expressed on nearly all monocytes, unlike previously thought. Glucocorticoids and IL-10 induce CD163 expression on monocytes through independent pathways.

Area of Science:

  • Immunology
  • Cell Biology

Background:

  • CD163, a scavenger receptor, is known to be expressed on macrophages.
  • Previous studies reported low expression on peripheral blood monocytes (<50%).

Purpose of the Study:

  • To clarify the expression pattern of CD163 on human monocytes and monocyte-derived cells.
  • To investigate the regulatory mechanisms of CD163 expression, including the roles of glucocorticoids and IL-10.
  • To determine if the monocyte marker p155 is identical to CD163.

Main Methods:

  • Flow cytometry analysis of peripheral blood monocytes and monocyte-derived dendritic cells.
  • Culture of human monocytes with glucocorticoids and cytokines (IL-10, IL-4, IL-13).
  • Western blot and flow cytometry on HEK 293 cells transfected with CD163 cDNA, using antibodies for CD163 and p155.

Main Results:

  • >99% of CD14+ monocytes express CD163; monocyte-derived dendritic cells show low expression.
  • Interleukin-10 (IL-10) and glucocorticoids induce high CD163 expression on cultured monocytes.
  • Glucocorticoid-induced CD163 expression is IL-10 independent and additive with IL-10.
  • IL-4 and IL-13 do not increase CD163 expression.
  • The monocyte/macrophage marker p155 is confirmed to be identical to CD163.

Conclusions:

  • CD163 is ubiquitously expressed on human monocytes, revising previous understanding.
  • Glucocorticoids and IL-10 are key inducers of monocyte CD163 expression via distinct pathways.
  • The identity of p155 as CD163 provides functional context for this scavenger receptor on monocytes.

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