Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Shiga Toxin-Producing Escherichia coli.

Jaeger1, Acheson

  • 1Division of Geographic Medicine and Infectious Diseases, New England Medical Center, 750 Washington Street, Boston, MA 02111, USA. david.acheson@es.nemc.org

Current Infectious Disease Reports
|November 30, 2000
PubMed
Summary
This summary is machine-generated.

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

[In Process Citation]

Arzneimittel-Forschung·2000
Same author

Apoptosis and anti-apoptosis in oestrogen-receptor negative endometrial cancer cells in response to anastrozole, 4-hydroxytamoxifen and medroxyprogesterone acetate

European journal of cancer (Oxford, England : 1990)·2000
Same author

Memory effects in granular materials

Physical review letters·2000
Same author

Vortex flow and transverse flux screening at the bose glass transition

Physical review letters·2000
Same author

Tecto-RNA: One-Dimensional Self-Assembly through Tertiary Interactions This work was carried out in Strasbourg with the support of grants to N.B.L. from the NIH (1R15 GM55898) and the NIH Fogarty Institute (1-F06-TW02251-01) and the support of the CNRS to L.J. The authors wish to thank Eric Westhof for his support and encouragement of this work.

Angewandte Chemie (International ed. in English)·2000
Same author

Signatures of granular microstructure in dense shear flows

Nature·2000
Same journal

Chagas Disease: An Opportunistic Infection in AIDS.

Current infectious disease reports·2026
Same journal

Area-based Deprivation Indices and Healthcare-Associated Infections: A Narrative Review of Evidence.

Current infectious disease reports·2025
Same journal

Next Steps: Studying Diabetic Foot Infections with Next-Generation Molecular Assays.

Current infectious disease reports·2024
Same journal

To Tweet or Not to Tweet-a Review of the Viral Power of Twitter for Infectious Diseases.

Current infectious disease reports·2024
Same journal

Web alert.

Current infectious disease reports·2024
Same journal

Bacterial Vaginosis in Postmenopausal Women.

Current infectious disease reports·2023
See all related articles

Shiga toxin-producing Escherichia coli (STEC) cause significant foodborne illness outbreaks. Advances in molecular detection and surveillance aid in identifying risk factors for STEC infection, crucial for prevention due to limited treatments.

Area of Science:

  • Foodborne infectious diseases
  • Microbiology
  • Public health

Background:

  • Shiga toxin-producing Escherichia coli (STEC) are a growing concern for foodborne infectious diseases globally.
  • Centralized food processing and distribution in the U.S. contribute to multistate outbreaks of E. coli O157 and non-O157 serogroups.
  • Limited effective treatments necessitate a focus on prevention strategies.

Purpose of the Study:

  • To review recent developments in the epidemiology, pathogenesis, diagnosis, treatment, and prevention of STEC disease.
  • To highlight advancements in molecular detection and surveillance for identifying STEC outbreaks.
  • To underscore the importance of identifying risk factors for STEC infection.

Main Methods:

  • Literature review of recent publications on STEC disease.

Related Experiment Videos

  • Analysis of developments in molecular detection techniques.
  • Examination of active surveillance practices for STEC.
  • Main Results:

    • Molecular detection and surveillance have improved the recognition of STEC clonality and outbreaks.
    • Key risk factors for sporadic and outbreak STEC infections have been identified.
    • Progress has been made in understanding STEC epidemiology and pathogenesis.

    Conclusions:

    • Enhanced surveillance and molecular tools are vital for controlling STEC outbreaks.
    • Identifying risk factors is critical for developing effective prevention strategies against STEC.
    • Continued research is needed to improve diagnosis and treatment options for STEC infections.