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Failure to demonstrate peri-infarct depolarizations by repetitive MR diffusion imaging in acute human stroke

T Back1, J G Hirsch, K Szabo

  • 1Department of Neurology, Klinikum Mannheim, Ruprecht Karls University Heidelberg, Germany. Tobias.Back@lrz.uni-muenchen.de

Stroke
|January 11, 2000
PubMed
Abstract

Insights

Peri-infarct depolarizations (PIDs) were not detected in human stroke patients using advanced MRI. This suggests that PIDs, observed in animal models, may not play a significant role in early human brain infarct enlargement.

Area of Science:

  • Neuroscience
  • Radiology
  • Stroke research

Background:

  • Peri-infarct depolarizations (PIDs) are linked to ischemic injury in experimental stroke models.
  • Diffusion-weighted MRI (DWI) has previously demonstrated PIDs in animal studies.

Purpose of the Study:

  • To investigate the relevance of PIDs in the early enlargement of human brain infarcts.
  • To test the hypothesis that PIDs contribute to infarct progression in humans.

Main Methods:

  • Ten human stroke patients underwent serial diffusion-weighted MRI (DWI) with high temporal resolution.
  • Apparent diffusion coefficient (ADC) maps were analyzed for spatial and temporal changes in infarcting brain tissue.

Main Results:

  • No time-dependent ADC changes indicative of PIDs were observed in human stroke patients.
  • ADC values significantly decreased from the infarct border to the infarct core, indicating progressive cytotoxic edema.

Conclusions:

  • The study failed to demonstrate PIDs in human stroke using a high-resolution MRI protocol.
  • Differences in PID detectability may exist between experimental stroke models and human stroke.

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