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Pneumococcal acute otitis media in children

G Kouppari1, A Zaphiropoulou, G Stamos

  • 1Microbiology Laboratory, Medical School, University of Athens, M. Asias 75, Goudi, Athens 11527, Greece.

Insights

Penicillin resistance in Streptococcus pneumoniae causing childhood otitis media was 15.9%. Multiresistant strains, often linked to specific serogroups, highlight the need for ongoing surveillance of antibiotic resistance in acute otitis media.

Area of Science:

  • Pediatric Infectious Diseases
  • Microbiology
  • Antimicrobial Resistance

Background:

  • Acute otitis media (AOM) is a common childhood infection.
  • Streptococcus pneumoniae is a leading bacterial cause of AOM.
  • Antibiotic resistance in S. pneumoniae poses a significant public health challenge.

Purpose of the Study:

  • To assess the prevalence of penicillin resistance in Streptococcus pneumoniae isolates from children with AOM.
  • To identify the antibiotic susceptibility patterns and serogroups of these isolates.
  • To evaluate the clinical and laboratory findings associated with S. pneumoniae AOM.

Main Methods:

  • Prospective study of 113 children with S. pneumoniae AOM from October 1995 to September 1996.
  • Antibiotic susceptibility testing using Kirby-Bauer and E-test methods.
  • Serotyping of isolates performed via Quellung reaction.

Main Results:

  • 15.9% of S. pneumoniae isolates showed penicillin insensitivity (4.4% highly resistant, 11.5% intermediate resistance).
  • 22.1% of isolates were resistant to one or more antibiotics; multiresistant strains were more common in penicillin-resistant isolates.
  • Predominant serogroups among penicillin-insensitive isolates included 19, 9, 23, 6, and 14.

Conclusions:

  • A significant proportion of S. pneumoniae causing AOM in children exhibited penicillin resistance.
  • Multiresistant S. pneumoniae isolates were frequently associated with specific serogroups (19, 9, 6, 23).
  • These findings underscore the importance of monitoring antibiotic resistance trends in pediatric AOM pathogens.
Abstract

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