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A novel MaxiK splice variant exhibits dominant-negative properties for surface expression
1Departments of Anesthesiology, Physiology, and Molecular and Medical Pharmacology and the Brain Research Institute, UCLA, Los Angeles, California 90095-7115, USA.
The Journal of Biological Chemistry
|March 30, 2001
Summary
A novel MaxiK channel splice variant (SV1) retains subunits in the endoplasmic reticulum, preventing functional MaxiK channel expression. This discovery reveals a new mechanism for regulating MaxiK channel activity.
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2026-06-19T13:41:22.049318+00:00