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Vitamin B12 treatment normalizes metabolic markers but has limited clinical effect: a randomized placebo-controlled

A M Hvas1, J Ellegaard, E Nexø

  • 1Department of Hematology, AAS, Aarhus University Hospital, Tage Hansens Gade 2, 8000 Aarhus C, Denmark. am.hvas@dadlnet.dk

Clinical Chemistry
|July 27, 2001
PubMed
Abstract

Insights

Vitamin B12 treatment effectively lowers elevated plasma methylmalonic acid (P-MMA) and homocysteine levels. However, the study found limited short-term clinical benefits for individuals with mild to modest P-MMA increases.

Area of Science:

  • Biochemistry
  • Clinical Nutrition
  • Neurology

Background:

  • The clinical relevance of elevated plasma methylmalonic acid (P-MMA) is not well-established.
  • Assessing the impact of vitamin B12 on P-MMA and related clinical outcomes is crucial.

Purpose of the Study:

  • To evaluate the effectiveness of vitamin B12 in reducing P-MMA and plasma total homocysteine.
  • To compare the biochemical changes with clinical benefits in patients with elevated P-MMA.

Main Methods:

  • A randomized, placebo-controlled study involving 140 participants with mildly to modestly increased P-MMA.
  • Baseline and 3-month assessments included medical history, laboratory tests, and a neurological disability score.

Main Results:

  • Vitamin B12 treatment significantly reduced P-MMA and plasma total homocysteine compared to placebo.
  • No significant differences were observed in hemoglobin, mean cell volume, or various symptom scores between groups.

Conclusions:

  • Vitamin B12 effectively lowers P-MMA and plasma total homocysteine.
  • Clinical benefits from vitamin B12 treatment appear limited in the short term for individuals with mild to modest P-MMA elevations.

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