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Optic nerve head behavior in Posner-Schlossman syndrome
V Darchuk1, J Sampaolesi, L Mato
1Department of Ophthalmology, School of Medicine, University of Buenos Aires, Fundación Argentina, Oftalmológica Juan Sampaolesi and Instituto de la Visión, Argentina.
International Ophthalmology
|April 12, 2002
Summary
Glaucomatocyclitic crisis causes temporary optic nerve head changes but no permanent damage. Surgery for Posner-Schlossman syndrome is reserved for severe cases or those with progressive optic neuropathy.
Area of Science:
- Ophthalmology
- Glaucoma Research
- Optic Nerve Studies
Background:
- Posner-Schlossman syndrome is characterized by recurrent episodes of unilateral acute anterior uveitis and transient ocular hypertension.
- Understanding the impact of these episodes on the optic nerve head is crucial for managing the condition.
Purpose of the Study:
- To analyze the effects of glaucomatocyclitic crisis on the optic nerve head's structure and hemodynamics.
- To evaluate surgical indications for Posner-Schlossman syndrome.
Main Methods:
- Four cases of Posner-Schlossman syndrome were studied.
- Retinal confocal tomography and Scanning Laser Doppler Flowmetry were used to assess optic disc phenomena.
- Automated computerized perimetry evaluated visual function correlation.
Main Results:
- Significant transient morphologic and hemodynamic changes in the optic nerve head were observed during ocular hypertension attacks.
- No permanent optic nerve head damage was found, except in a case associated with traumatic glaucoma.
- All parameters returned to normal after intraocular pressure remission.
Conclusions:
- Glaucomatocyclitic crises induce temporary optic nerve head alterations that do not cause lasting damage.
- Surgery is indicated for severe, disabling symptoms or progressive optic neuropathy with visual field loss, especially when associated with glaucoma.