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The Use of Probiotics in Head and Neck Infections

Kristian Roos1, Stig Holm

  • 1ENT Department, Lundby Hospital, Wieselgrensplatesen 2A, 41717 Göteborg, Sweden. Kristian.roos@lundbysjukhus.se

Insights

Normal upper respiratory tract bacteria prevent pathogen growth and recurrent infections. Restoring these beneficial bacteria, like alpha-streptococci, significantly reduces infection recurrences by outcompeting pathogens.

Area of Science:

  • Microbiology
  • Immunology
  • Infectious Diseases

Background:

  • Recurrent upper respiratory tract infections (URTIs) are common but poorly understood.
  • The role of normal bacterial flora in preventing pathogen colonization and reinfection is a key area of research.
  • Disruption of this protective flora is linked to increased infection frequency.

Purpose of the Study:

  • To investigate the protective mechanisms of normal respiratory tract bacteria against pathogens.
  • To evaluate the efficacy of recolonization with interfering bacteria in reducing recurrent URTIs.
  • To understand the interactions between normal flora, pathogens, and the host immune system.

Main Methods:

  • In vitro and in vivo studies examining bacterial adherence and growth inhibition.
  • Clinical application of recolonization with alpha-streptococci in patients with recurrent infections.
  • Analysis of proposed mechanisms including competition for adhesion sites and production of inhibitory substances.

Main Results:

  • Normal aerobic and anaerobic bacteria of the upper respiratory tract inhibit pathogen growth and colonization.
  • Recolonization with alpha-streptococci successfully reduced recurrence rates in acute streptococcal pharyngotonsillitis and acute otitis media.
  • Proposed mechanisms include competition for epithelial binding sites, hydrogen peroxide production, nutrient competition, and bacteriocin production.

Conclusions:

  • The normal bacterial flora of the upper respiratory tract plays a crucial role in preventing recurrent infections.
  • Alpha-streptococci are effective in reducing URTI recurrences through mechanisms like bacterial interference.
  • Further research into host-microbe interactions and immune components like lactoferrin is important for understanding URTI pathogenesis.

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