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Plasmid mediated antibiotic resistance in Klebsiella pneumoniae

R Misra1, M Kumar, P K Menon

  • 1Department of Microbiology, Armed Forces Medical College, Pune.

Insights

Klebsiella pneumoniae isolates show high antibiotic resistance, with over 40% exhibiting resistance to 9-12 drugs. Plasmid-mediated resistance transfer was confirmed, highlighting a significant public health concern.

Area of Science:

  • Medical Microbiology
  • Infectious Diseases
  • Antimicrobial Resistance

Background:

  • Klebsiella pneumoniae is a significant opportunistic pathogen in healthcare settings.
  • Antibiotic resistance in K. pneumoniae poses a major threat to patient treatment and outcomes.
  • Understanding resistance mechanisms is crucial for effective infection control.

Purpose of the Study:

  • To investigate the antibiotic resistance patterns of Klebsiella pneumoniae isolates from hospitalized patients.
  • To determine the prevalence of multidrug resistance (MDR) in these isolates.
  • To explore the role of plasmids in the transfer of antibiotic resistance.

Main Methods:

  • Isolation and identification of Klebsiella pneumoniae from clinical samples (urine, pus, blood, sputum).
  • Antimicrobial susceptibility testing against a panel of 12 antibiotics.
  • Plasmid profiling using agarose gel electrophoresis and conjugation experiments to assess resistance transfer.

Main Results:

  • High resistance rates observed for ampicillin (93.3%), sulphamethoxazole (96%), and trimethoprim (94%).
  • A significant proportion of strains (40.4%) displayed resistance to 9-12 antibiotics, indicating extensive multidrug resistance.
  • Conjugation experiments confirmed the transfer of resistance genes, with a large plasmid (98.7 kb) identified in resistant strains.

Conclusions:

  • Klebsiella pneumoniae isolates from tertiary care hospitals exhibit alarming levels of antibiotic resistance.
  • Plasmid-mediated horizontal gene transfer contributes to the dissemination of MDR in K. pneumoniae.
  • Urgent strategies are needed to combat the spread of resistant strains and preserve antibiotic efficacy.

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