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Effects of NFkappaB decoy oligonucleotides released from biodegradable polymer microparticles on a glioblastoma cell

Jagjit S Gill1, Xun Zhu, Michael J Moore

  • 1Molecular Neuroscience Program, Mayo Clinic, Rochester, MN 55905, USA.

Biomaterials
|June 13, 2002
PubMed

Insights

A nuclear factor-kappa B (NF-kappaB) decoy oligonucleotide strategy significantly reduced glioblastoma cell growth by inhibiting cell cycle progression. Poly(DL-lactic-co-glycolic acid) microparticles effectively delivered these decoy oligonucleotides, showing therapeutic potential.

Area of Science:

  • Oncology
  • Molecular Biology
  • Biotechnology

Background:

  • Nuclear factor-kappa B (NF-kappaB) is aberrantly expressed in glioblastoma (GBM) cell lines and drives their growth.
  • NF-kappaB activation is crucial for regulating GBM cell proliferation and cell cycle progression.
  • Untransformed glial cells do not exhibit the same level of NF-kappaB activity as GBM cells.

Purpose of the Study:

  • To investigate the efficacy of a nuclear factor-kappa B (NF-kappaB) decoy oligonucleotide (ODN) strategy in inhibiting glioblastoma (GBM) cell growth.
  • To evaluate the use of poly(DL-lactic-co-glycolic acid) (PLGA) microparticles as a delivery system for NF-kappaB decoy ODNs in vitro.
  • To elucidate the mechanism by which NF-kappaB influences GBM cell cycle regulation.

Main Methods:

  • Utilized NF-kappaB decoy oligonucleotides designed to inhibit nuclear translocation of NF-kappaB.
  • Encapsulated decoy ODNs within PLGA microparticles for controlled release.
  • Assessed cell number, cyclin D1 protein expression, and Cdk-4 activity in GBM cell lines.
  • Monitored ODN release kinetics from PLGA microparticles in phosphate-buffered saline.

Main Results:

  • NF-kappaB decoy ODNs significantly reduced GBM cell number by up to 45% compared to controls.
  • The reduction in cell number correlated with decreased cyclin D1 protein expression and Cdk-4 activity.
  • PLGA microparticles demonstrated a 66% yield for ODN encapsulation and sustained release of active ODNs for up to 28 days.
  • Released decoy ODNs from PLGA microparticles retained biological activity, significantly reducing GBM cell growth in vitro.

Conclusions:

  • NF-kappaB plays a critical role in mediating GBM cell cycle progression and growth.
  • The NF-kappaB decoy ODN strategy presents a potential therapeutic approach for controlling human GBM cell growth.
  • PLGA microparticles are suitable delivery vehicles for implementing the decoy ODN strategy against GBM in vitro.

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